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| Osteoporosis will remain an important theme on the EFORT agenda because of its increasing significance, Prof Puhl emphasized. For the next Congress in Madrid in 2010 we are planning, together with patient organizations, a debate with political decision makers on the European level. For the sensitizing of the public and policy makers about this disease will be a decisive factor in finally providing care corresponding to the current medical knowledge to those suffering from it. EFORT Osteoporosis, or bone loss, is a disease that is most common in the elderly population, affecting women more often than men. Until now, it was not clear exactly how the disease develops. Researchers of the Max Delbruck Center for Molecular Medicine (MDC) Berlin-Buch, Germany, have now elucidated a molecular mechanism which regulates the equilibrium between bone formation and bone resorption. Valerie Begay, and Professor Achim Leutz were able to show that two different forms of a gene switch - a short isoform and a long isoform - determine this process. The MDC researchers hope these findings will lead to new therapies for this bone disease.
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The bones lose their density and are therefore prone to breakage. Even minor falls can lead to serious bone fractures. The interplay between two cell types determines bone density: bone forming cells (osteoblasts) and bone resorbing cells (osteoclasts). The equilibrium between these two cell types is strictly regulated to prevent the formation of either too much or too little bone. Begay, and Professor Leutz have now elucidated a complicated mechanism which maintains the equilibrium between bone formation and bone resorption.
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